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Research Article

The FGFR4-G388R Polymorphism Promotes Mitochondrial STAT3 Serine Phosphorylation to Facilitate Pituitary Growth Hormone Cell Tumorigenesis

  • Toru Tateno,

    Affiliations: Ontario Cancer Institute, University Health Network, Toronto, Canada, The Endocrine Oncology Site Group, Princess Margaret Hospital, Toronto, Canada, Department of Medicine, University of Toronto, Toronto, Canada

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  • Sylvia L. Asa,

    Affiliations: Ontario Cancer Institute, University Health Network, Toronto, Canada, The Endocrine Oncology Site Group, Princess Margaret Hospital, Toronto, Canada, Department of Laboratory Medicine and Pathobiology, University of Toronto, Toronto, Canada

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  • Lei Zheng,

    Affiliations: Ontario Cancer Institute, University Health Network, Toronto, Canada, The Endocrine Oncology Site Group, Princess Margaret Hospital, Toronto, Canada, Department of Medicine, University of Toronto, Toronto, Canada

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  • Thomas Mayr,

    Affiliation: Department of Molecular Biology, Max-Planck-Institute of Biochemistry, Martinsried, Germany

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  • Axel Ullrich,

    Affiliation: Department of Molecular Biology, Max-Planck-Institute of Biochemistry, Martinsried, Germany

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  • Shereen Ezzat mail

    shereen.ezzat@utoronto.ca

    Affiliations: Ontario Cancer Institute, University Health Network, Toronto, Canada, The Endocrine Oncology Site Group, Princess Margaret Hospital, Toronto, Canada, Department of Medicine, University of Toronto, Toronto, Canada

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  • Published: December 08, 2011
  • DOI: 10.1371/journal.pgen.1002400

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